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Recently, metagenomic analysis revealed fungal DNA in tumors and suggested their role in inflammation-driven carcinogenesis [20]

This sophisticated delivery mechanism transforms a notoriously poorly absorbed nutrient into a highly bioavailable therapeutic tool, capable of significantly elevating systemic bodily stores of this crucial antioxidant

Results from the PIVENS trial showed that oral administration of the anti-oxidant vitamin E improved liver dysfunction and the pathological conditions of NASH [17]

eNOS uncoupling is caused by deficiency or oxidation of tetrahydrobiopterin, an important cofactor for eNOS action, leading to increased generation of superoxide and decreased NO bioavailability (155)